KNES 466 Lecture Notes - Lecture 3: Cardiac Muscle, Skeletal Muscle, Cardiac Output

• School: University of Maryland
• Department: Kinesiology
• Course: KNES 466
• Professor: marc Rogers

Document Summary

Actin(thin)/myosin(thick) filaments, creates movement in power stroke, condensing cell and producing force. Transverse tubule--facilitates entry of action potential, which travels surface of cardiac cell and enters via t-tubule and go into cell: has protein receptor which facilitates release of calcium. Sarcoplasmic reticulum-primary function is to store, release, reuptake calcium, which is essential for contraction of cardiac cells. Difference between cardiac and skeletal: cardiac-two receptors for proteins which facilitate release of calcium. Important because cardiac cells are vital for every function, number of redundant mechanisms which contribute to maintenance of cardiac output: cardiac-high concentration of mitochondria in cardiac muscle. Side by side image analysis of cardiac vs muscle would be based on mitochondria. Cardiac cells rely on aerobic energy processes, causing necessity for many mitochondria. Syncytium--ability for all the hearts cells to contract at the same time. Aerobic exercise training persons, skeletal muscle begins to resemble cardiac muscle to a greater extent. Increase in oxidative enzymes, size of mitochondria, etc.