BCS 242 Lecture Notes - Lecture 11: Partial Seizure, Absence Seizure, Status Epilepticus
12 Oct 2016
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Document Summary
Epilepsy: low-voltage, mixed frequencies, pseudorandom/chaotic patterns complex signals carrying info for behavior. Seizure: excessive excitation failure of inhibitory mechanisms high-voltage activity spreading thru networks: complex signaling gets degraded behavior becomes driven, stereotyped, unresponsive to environment, self-limited, onset. Spread of paroxysmal electric activity partial seizure: generalized seizure: starting from all areas of brain at once (ie. , absence seizure) Interactions b/w thalamus & throughout neocortex spreads. Primary: diffuse onset can start in subcortex/neocortex. Show other seizure symptoms before onset: partial. Simple: seizure starts focally, stays focal sensory/motor effects. Complex: awareness, memory impaired impairment level depends on onset & spread pattern. Aura: warning" that can occur before seizure onset (ie. , vision, d j vu) Abnormal networks become set up seizure activity: cryptogenic: small, developmental abnormalities (eg. , tumors) Causes of cog/behavioral dysfunction: ictal activity: during seizure stereotyped, driven, unconsciously constrolled, post-ictal: confusion, aggression, fatigue brain reboots".
