PSYC 3230 Lecture Notes - Lecture 2: Primary Care Physician, Electrodermal Activity, Anxiety Disorder
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Ge(cid:374)eti(cid:272) (cid:448)ul(cid:374)era(cid:271)ilit(cid:455) does(cid:374)"t (cid:272)ause a(cid:374)(cid:454)iet(cid:455)/pa(cid:374)i(cid:272) dire(cid:272)tl(cid:455); (cid:373)ust also (cid:271)e e(cid:454)posed to the right environmental stressors/conditions. When stress response is chronically activated, certain genes (cid:272)a(cid:374) (cid:271)e (cid:862)tur(cid:374)ed o(cid:374)(cid:863) (epigenetics) and contribute to anxiety) Brain stem (primitive area of brain; monitors changes in body functions) Corte(cid:454) (cid:894)outer part of (cid:271)rai(cid:374) that (cid:862)(cid:373)akes us hu(cid:373)a(cid:374)(cid:863)(cid:895) Act as mediator between brain stem and cortex. Systems that are activated by signals from brainstem limbic system cortex. Classical and operant conditioning- symptoms are a result of learned associations. Biological and psychological vulnerabilities are triggered by stressful life events (divorce, difficulties at work, pressure to excel in school, death of loved one) Some people may be more likely than others to respond to stress with anxiety. Disorders that share features of excessive fear and anxiety and related behavioral disturbance (ex: avoidance) Autonomic restrictors: less responsiveness on physiological measures (heart rate, skin conductance) than those with other anxiety disorders.