Global Health and Health Policy 50 Lecture 1: Coagulation Modifier Drugs 241

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thestudygeek

2 Nov 2021

School

Harvard University

Department

Global Health and Health Policy

Course

Global Health and Health Policy 50

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10. 10. 2018: general term for any process that stops bleeding, coagulation is hemostasis that occurs because of the physiologic clotting of blood, complex relationship between substances that promote clot formation and either inhibit coagulation or. Ashley hawthorne dissolve a formed clot: thrombus: technical term for a blood clot, embolus: thrombus that moves through blood vessels. Cascade : each activated factor serves as a catalyst that amplifies the next reaction, result is fibrin, a clot-forming substance fibrinolytic system is activated (initiates the breakdown of clots and balances the clotting process) Intrinsic pathway and extrinsic pathway: these pathways are activated by different types of injury, extrinsic: blood vessels damaged by the penetration from the outside (knife or bullet wound) Intrinsic: the components of the intrinsic pathway are present in the blood in their inactive forms, activated when in contact with exposed collagen on the inside of damaged blood vessels.

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Briefly describe how death receptors trigger the extrinsic apoptosis pathway through the formation of a death-inducing signaling complex (DISC). Be sure to include: (1) What the ligand is, where it comes from and what it binds, (2) the intracellular cascade that produces the DISC, and (3) the type of caspase that is activated and how it functions.

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Integral membrane proteins that exhibit an intrinsic activity of hydrolyzing ATP to ADP and Pi most likely participate in Question 6 options: A. binding polypeptide hormones. B. generating intracellular messengers during signal transduction. C. initiating one or more steps in blood coagulation. D. moving cations against their concentration gradient. E. targeting proteins to different organelles.

1. Be able to define the following terms: releasing hormone,inhibiting hornone, trooic hormone, nontropic hormones,neurosecretory cells.

2. What are hormones?

3. What are two specific differences between the endocrine andnervous systems?

4. What are the four different types of cell signaling?

5. What are the three types of hormonal control pathways? Givean example of each.

6. What is local regulator?

7. What type of feedback is a common feature of pathways thatcontrol homeostasis?

8. What are the three major classes of molecules that functionas hormones? Which are water-souble and which are lipid-souble?

9. Signaling by a hormone involves three key events. What arethey?

10. What is the correlation between solubility of a hormone andthe location of its receptors?

11. In addition to receptor location, water-soluble andlipid-soluble hormones also exhibit additional differences in theirresponse pathways. What are these differences?

12. What is signal transduction pathway?

13. Why can the same hormone have different effects on differentbody tissues?

14. Liver cells and the blood vessels of skeletal muscle cellshave identical surface receptors for epinephrine, yet the responeof these two cells types is dramatically different. Explain howthis is possible.

15. In the "fight or fight response" the release of epinephrinefrom the adrenal medulla cause skeletal muscle blood vessels todilate while simultaneously constricting blood vessels of theintestine. Explain how this is possible.

16. List the three types of local regulators discussed in classand explain their function.

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Global Health and Health Policy 50 Lecture 1: Coagulation Modifier Drugs 241

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