ANP 1105 Chapter Notes -Hemoglobin, Erythropoietin, Coagulation

• School: University of Ottawa
• Department: Anatomy and Physiology
• Course: ANP 1105
• Professor: Jacqueline Carnegie

Document Summary

Antibodies, complement, wbcs. (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) Reticulocyte: doesn"t have a complete shape, still contains ribosomes and these will disintegrate. (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) Loss of fe: feces, urine, sweat. (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) Platelet formation is regulated by thrombopoietin, responsible for enough platelets produced liver. in (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) 3 phases: prothrombin activator formed, prothrombin to throbin, fibrinogen molecules (cid:224) fibrin mesh. (cid:222) many procoagulants + anticoagulants; latter predominate in intact vessel. 4. 1. 3. 3: intrinsic and extrinsic pathways for the formation of prothrombin activator. (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) (cid:222) Intrinsic: clotting of blood outside the body, everything it needs is present, slower, opportunity to stop blood clotting, trigger: exposure to collagen, greater capacity.