PSYC 446 Lecture Notes - Lecture 4: Hypothalamus, Prospective Memory, Sympathetic Nervous System
18 Sep 2018
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Why do we age: programmed theories cellular clock theories, hormonal stress theory, damage theories free-radical theory, video about stress theory telomere they shorten with chronic stress. Free-radical theory: mito(cid:272)ho(cid:374)d(cid:396)ial a(cid:271)ilit(cid:455) to (cid:396)epai(cid:396) dna da(cid:373)age (cid:449)ith age de(cid:272)(cid:396)eased (cid:272)ell effi(cid:272)ie(cid:374)(cid:272)(cid:455, too (cid:373)u(cid:272)h (cid:373)ito(cid:272)ho(cid:374)d(cid:396)ial da(cid:373)age (cid:272)ell death, o(cid:454)idati(cid:448)e da(cid:373)age f(cid:396)ee (cid:396)adi(cid:272)als dna da(cid:373)age, (cid:272)(cid:396)oss-linking of proteins, formation of age pigments. Injury caused by free radicals initiates a self-perpetuating cycle in which oxidative damage impairs mitochondrial function, which results in the generation of even greater amounts of oxygen-free radicals. The more we grow older, the lesser the genetic factors are important, and the more our environmental factors come at play. The frailty of old age is essentially avoidable and largely reversible. Definition: memory refers to the storage, retention and recall of information including past experiences, knowledge and thoughts: only some types of memory loss are associated with normal aging, other types are typical of disease states.
