PSYC 211 Lecture Notes - Lecture 4: Adenosine Receptor, Sleep Paralysis, Transcranial Magnetic Stimulation
14 Sep 2016
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Lecture 4 end of psychopharmacology & experimental methods & vision. Chapter 4 psychopharmacology: acetylcholine: muscular movement, m(cid:455)asthe(cid:374)ia g(cid:396)a(cid:448)is: a he(cid:396)edita(cid:396)(cid:455) diso(cid:396)de(cid:396) i(cid:374) (cid:449)hi(cid:272)h the pe(cid:396)so(cid:374)(cid:859)s o(cid:449)(cid:374) i(cid:373)(cid:373)u(cid:374)e system attacks healthy acetylcholine receptors. People with this disorder become noticeably weaker and weaker over time (fatigability). We do(cid:374)(cid:859)t (cid:455)et ha(cid:448)e a good way to give these people back functional receptors, but with drugs like. Neostigmine we can make the released acetylcholine stay around for longer periods of time: cocaine, crystal meth, antipsychotics (neuroleptics) Outline: experimental ablation (histology: (cid:13559) , recording and stimulating neural activity, neurochemical methods, genetic methods. Dest(cid:396)o(cid:455)i(cid:374)g pa(cid:396)t of the (cid:271)(cid:396)ai(cid:374) a(cid:374)d e(cid:448)aluati(cid:374)g the a(cid:374)i(cid:373)al(cid:859)s su(cid:271)se(cid:395)ue(cid:374)t (cid:271)eha(cid:448)io(cid:396). Not involve in removing brain tissue; but destroys some tissue and leaves it in place. Producing brain lesions: radio frequency (rf) lesions: destroy subcortical regions (regions located beneath the cortex) by radio frequency current, produced by passing electrical current through a metal wire and burn the region of brain tissue.
