ANSC 2340 Lecture Notes - Lecture 21: Lactic Acid, Excess Post-Exercise Oxygen Consumption, Exsanguination
17 Apr 2016
School
Department
Course
Professor
Document Summary
The inability of a muscle to contract after prolonged activity. Relative deficit of atp: not all muscle fibers are able to contract. Muscle cell has a hard time repolarizing, and therefore you get an extended refractory time and the muscle cannot respond to any more stimuli. Lactic acid: blood supply to the muscle is limited, conversion from aerobic to anaerobic. Oxygen debt: oxygen is necessary for aerobic metabolism to produce atp. The increase in lactic acid triggers an increase in respiration. Electromyogram: measures the electrical activity in the muscles. Muscles lose: oxygen nutrients heat removal: 75% of atp energy is released as heat, the blood usually removes this heat but not after the animal is dead. Waste removal: no blood flow means no waste removal. Anaerobic metabolism: any oxygen in the myoglobin is used up very quickly biochemical reactions still continue but it is through glycolysis generation of atp tries to continue because it does not need nervous system input.
