PATH 3610 Lecture Notes - Lecture 5: Cyclin-Dependent Kinase Inhibitor 1C, Cyclin-Dependent Kinase 2, Cyclin-Dependent Kinase 1
3 Feb 2016
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Document Summary
Extensive injury to nervous or cardiac muscle tissue will lead to a functional deficit: the primary motivators for cell replication are proteins called cyclins, which interact with cyclin dependent kinases within the nucleus that control the entry and progression of cells through the cell cycle, rate of cyclin activity can be further modified by a variety of factors: Develop plans for surveillance for future disease outbreaks: some pathogens will either specifically target the fast dividing crypt stem cells (i. e. canine parvovirus) or will cause nonspecific injury to all levels of the intestinal mucosa (i. e. profound necrosis associated with salmonella spp. ) Healing and the permanent cell population: case study: 42 year old man has acute chest pain and difficult breathing, a myocardial infarction is diagnosed, if the patient survives the attack, the myocardium will heal with scar formation without any significant regeneration, factors that will influence the prognosis for this patient will be age, etc.
