PHAR 201 Lecture Notes - Lecture 13: Anxiolytic, Confusion, Gluconeogenesis
16 Dec 2015
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In science it"s a class of compounds, whereas in society, it is a specific drug (ethanol) Impaired motor skills/ataxia (full loss of bodily movement control) Which receptors get simulated, and what role does this have on. Increases fluidity by altering membrane phospholipid order: this causes cns depression (the mechanism is like a general anesthetic) Gabaa receptors get stimulated, enhancing their role in. Cns inhibition: recall that gaba lets cl- into the postsynaptic neuron, making it more negative and less excitable. Reduces effects/release of excitatory neurotransmitters like glutamate and serotonin calcium channels: direct receptor effects + inhibiting voltage-gated, stops ca2+ from entering axon terminal and fusing with neurotransmitter vesicles. This prevents their release into the synapses flush . Prominent in cutaneous (skin) blood vessels, drunkard"s. Adh normally increase permeability of kidney ducts to water, allowing water to flow back into the blood plasma. When inhibited, water is lost in large volume as urine.
