PSYC 101 Lecture Notes - Lecture 11: Neurodegeneration, Prefrontal Cortex, Glutamine

• School: UBC
• Department: Psychology
• Course: PSYC 101
• Professor: Steven Barnes, Grace Truong

Document Summary

Any kind of mental illnesses could be explained by these factors, and they all play a role in the existence/development of the mental illness. Neurons and in synapse between muscles and organs. Disorientation, problems with language, mood swings, loss of motivation. Motor behavior, motivation, pleasure and emotional arousal. Overactivity of dopamine receptors; high levels of dopamine. Progressive nervous system disorder that affects movement (resting tremor Decay of dopamine neurons, low levels of dopamine, less activity in basal ganglia. Overexcitation of neurons (too much glutamate or too little gaba ) Low levels of serotonin and norep associated w depression. Runner"s high (not only related to endorphins) Drugs that increase the action of a nt. Increase the production and release of nts. Bind to the receptors on the post-synaptic neurons: activation or increase the nt effect. Bind to autoreceptors and block their inhibitory effect. Drugs that block the function of a nt. Bind to and block the receptors on the post-synaptic neurons.