BCH 4123 Lecture Notes - Lecture 14: Ectopic Pregnancy, Follicular Phase, Fallopian Tube
6 Mar 2017
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Document Summary
Gnrh released in pulses: stimulates ap to produce fsh and lh, males produce testosterone, females produces estradiol. Negative feedback on both hypothalamus and ap. Granulosa cells respond to fsh and produce estrogens. Theca cells respond to lh and produce androgens. Implantation can occur and causes ectopic pregnancy: ciliated and lined with smooth muscle to propel egg to uterus. Uterus: endometrium responds to hormonal changes in menstrual cycle. Menstrual phase by low hormone levels: myometrium is muscle layer. Can cause abdominal pain: dilates during childbirth, cervical mucus. Barrier and transport medium for sperm depending on time of cycle. Vagina: glands produce lubrication during intercourse. Mammary glands: respond to prolactin during pregnancy. Enlarge and prepare for mild production: response to oxytocin causes milk letdown. Can vary in length from 10-16 days. Increased estrogens causes lh surge to causes ovulation: luteal phase. Reconstruction of endometrium after menstruation: secretory phase. Lose negative feedback to ap and hypothalamus so cycle starts again.
