BIOC34H3 Lecture Notes - Lecture 7: Heart Valve, Pulmonary Vein, Cardiac Output
Document Summary
Prevents it from maintaining a proper cardiac output. On a starling curve lower curve than normal. Will never reach the norm, even if there is sympathetic stimulation. Blockage in arteries via fatty deposits or placques. Stenosis : prevents opening of semilunar valves caused by long-term high bp causes thickening of muscles to compensate for the blockage of semilunar valves. Cardiac hypertrophy: thickening of ventricles because of it needs to bump blood out more. If the valves are blocked, the blood needs to be pumped out even more and over time hypertrophy thickening of the walls occurs. High bp will decrease edv, increase cardiac work, lose ventricular compliance decrease. Cyanosis: turning blue due to low o2. Ventricular pressure-volume loop: normal, chronic heart failure, acute heart failure. Heart failure = inefficient blood pumping = increase in esv where there is low volume of blood inventricles bc heart is incapable of pumping blood into ventricles.
