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Lecture 11

PSYC23H3 Lecture Notes - Lecture 11: Donald Winnicott, Developmental Psychobiology, Memory Consolidation


Department
Psychology
Course Code
PSYC23H3
Professor
David Haley
Lecture
11

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LECT 11
Recap
- Attachment model of Psychopathology
o Sleep’s regulatory role in daytime functioning is significant across the
lifespan get more sleep! and don’t ever forget about PEP!
o Elevated cortisol and sleep problems (e.g., night terrors) predicted
greater internalizing sysmptoms (e.g., anxiety and depression)
High cortisol + parasomnia internalizing symtoms
Clinical approach you don’t get them when their still fine and healthy
- you get them when they are full blown crazy
o but the developmental psychpathologists/biologists approach
contrasts from this
o they get to see the kids when they are still fine, and follow them
through until they get the disorder
- we don’t have many memories of our childhood
o even though we don’t remember it, theres a possibility that the
experience is still important
it still infl who you develop
o @ 6mo old you can have memory!!
The learning & memory is in place at that time
Even if you can remember it now, doesn’t mean you couldn’t
remember it when it was happening
- You cant look at parents & children separate
o They have to be looked at together
In the mid 20th c., Donald Winnicott and Melanie Klein used their seminal work with
infants and young children to formulate developmental models describing how the
mind comes into being.
- Winnicott said that there is no baby without a mother and no mother without
a baby (Winnicott 1960).
o He was saying that the mind comes into being from within the context
of a significant relationship, from within an intersubjective field. We
begin ourselves by being with another.
Memory
The first notion to get rid of is that memory is primarily or literally
reduplicative, or reproductive. In a world of constantly changing environment,
literal recall is extraordinarily unimportant and not the most ...memory
appears to be an affair of construction rather than reproduction.
Bartlett, 1932
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The first notion to get rid of is that memory is primarily or literally
reduplicative, or reproductive. In a world of constantly changing environment,
literal recall is extraordinarily unimportant and not the most ...memory
appears to be an affair of construction rather than reproduction.
Bartlett, 1932
- memory isn’t fixed
o its dynamic/changing
- every time you remember something, it gets recreated
- you need to be able to shape/construct you memory
o highlight the key points
o don’t get remember everything literally
Metaphors For Memory
- deal w the way in which memory is stored/formed
o when you are interesting in something, you heat up your memory
capacity
- we are constantly reconstructing and changing out stories
- heated wax
o you heat up the wax, and impression/experience can shape the wax a
certain way
o time element the idea of it being totally fixed is debated/questioned
the wax can be reheated & the shape can be changed
- emotional stimulation heats up wax (ie. someone slaps you in the face)
o thus helping you encode more details to further remember &
consolidate
Stress and Memory
- Long-term potentiation (LTP) in hippocampus
o LTP: neurons can fire for a long period of time
neuron keeps firing & responds to a particular stimulus
o rats w LPT had enhanced performance & better memory
- Stress receptors expressed in the hippocampus
o stress receptors can directly affect key parts of the brain that are
involved in memory
can shape our experiences
o stress receptors in hippocampus (-) feedback; can shut off HPA-axis
once cortisol enters brain
we know this
- Stress receptor occupation
o stress receptors in hippocampus involved in memory in general
amount of cortisol that reaches hippocampus, might have a
curvilinear relationship
too little not good memory consolidation
moderate good; enhances memory consolidation
too much disrupts memory consolidation
find more resources at oneclass.com
find more resources at oneclass.com

Only pages 1-3 are available for preview. Some parts have been intentionally blurred.

- Stress hormones and LTP
o too little cortisol no activation
o moderate activation
o too much no activation
*Moderate stress, not negative cascade effect of chronic stress*
Stress Hormones Form Blood
- moderate amount of cortisol enhances the process of connecting neurons
o you have to get the neurons fired & connected to other neurons so you
can create the wax that forms and supports a particular
impression/experience
Amygdala
- hippocampus coordinates w amygdala w fearful/sad experiences
- Patient S.M
o Bilateral amygdala lesions (lipoid proteinosis)
o Fearless
This isn’t always good; it can kill you
o Emotional but enjoys fearful stimuli
o Doesnt learn to form associations between context and scary events
They forget the emotional/fearful aspect
- Amygdala helps activate fear system
o All fearful experiences S.M always reports no/little fear
Three + Stages of Memory
- when you encode something perception (learning for the first time)
- over time, memory gets constructed; memory is forming
o consolidation;
the mystery is in consolidation/reconsolidation
- ability to retrieve depends on how you consolidated & encoded the memory
Fear Memories
- when you retrieve the memory, you invoke a new stage of consolidation
(reconsolidation)
o when you reconsolidate memory, you can change the memory slightly
o ie. ppl w PTSD, when they are asked to remember, they are req to
change the memory slightly so they don’t freak out
- memory consolidation when activated timely
o a few hours
find more resources at oneclass.com
find more resources at oneclass.com
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