PSL301H1 Lecture Notes - Lecture 13: Extracellular Fluid, Hydrostatics, Capillary Pressure

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5 Feb 2017
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PSL301H1 Full Course Notes
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Responses to exercise: capillaries, lymphatic system and edema. More blood goes back to the heart to get o2. Inc in sym activity, withdrawal of parasym activity. Neurotransmitter junctions in skeletal muscle send signals to the cardiovascular control center (ccc) Effects on hr + contractility, resistance arterioles in metabolically inactive tissues. The increase hr decreases the amount of blood in the ventricles in resp to the high venous return --> this protects the heart from overstretching. Sympathetic activity causes them to think higher pressures are normal. Local metabolites mediate profound vasodilation in skeletal muscle. Shown below is the tradeoffs the body makes to increase blood to muscle. However, map is known to elevate slightly during exercise. They must behave differently so they don"t normalize blood pressure. In regular conditions, the baroreceptors would panic to bring bp back down. We don"t know the mechanisms of this for sure. Given a resting hr and sv of 75 bpm and 80 ml, respectively.

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