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Why is this a false statement?

Antibody activation of complement proteins to form MAC is aninnate response to bacterial invasion and acts synergistically withcomplement proteins acting as opsonins

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Nestor Rutherford
Nestor RutherfordLv2
28 Sep 2019

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Related questions

Which of the following is a characteristic of the adaptive immune system?

a .It is a specific response against microbial antigen

b. It includes inflammation.

c. It activates the alternative complement pathway.

d. It includes defenses on the body surfaces.

2. C-reactive protein is an opsonin because it:

a. creates a channel in bacterial membranes

b. increases C3b binding to a microbe.

c. forms the pathogen-associated molecular patterns.

d. enhances phagocytosis.

3. The rapid increase in antibody production after repeated exposures to the same antigen is due to:

a. antibody-dependent cellular cytotoxicity.

b. higher levels of IgE

c. presence of memory cells.

d. clonal proliferation of NK cells

4. Cytotoxic T cells can kill cancer cells by:

a. inducing apoptosis with granzyme.b. release of membrane attack complex.

c. activate the production of complement C3b

d. phagocytosis of opsonized cells.

5. Which event is not required for the activation of helper T cells?

a. The presence of interleukin-1

b. Binding of CD8 receptors

c. Binding of matching non-antigenic proteins of the antigen-presenting cel

d. Presentation of an antigen bound to class II MHC

1.How do some pathogens prevent complement activation or avoid the effects of activated complement?

A. some pathogens hijack the host's regulatory membrane proteins that inactivate C3b, preventing the triggering of the alternative activation pathway AND some pathogens produce C5a peptidase that causes destruction of phagoctyes recruited to the area of infection

B. some pathogens produce C5a peptidase that causes destruction of phagoctyes recruited to the area of infection

C. some pathogens produce C5a peptidase that destroys complement C5a, a chemoattractant that recruits phagocytes to the area of infection

D. Some pathogen hijack the host's regultory membrane proteins that inactivate C3b, preventing the triggering of the alternative activation pathway AND some pathogens produce C5a peptidase that destroys complement C5a, a chemoattractant that recruits phagocytes to the area of infection.

2. One of the most important jobs of dendritic cells is

A. production of complement proteins

B. activation of T cells by breakdown of whole antigens and presentation of antigenic epitopes on MHC molecules

C. stimulation of cytokine production by neutrophils

D. activation of B cells by combining epitopes and presentation of whole antigens on MHC molecules

E. production of IgD

3. Normal Microbiota

A. play no role in affecting pathogen growth

B. are the organisms that typically reside on and in your body

C. enhance infection by pathogens

D. are the organisms that typically reside on and in your body AND protect aganist infection by pathogens

E. protect against infection by pathogens.

4. why is the process of gene rearrangement used in creating antibodies and T-cell receptors (TCRs) so important?

A. the given choices are all reasons that generearrangement is critical in adaptive immunity

B. This is how our systems produce memory of previous exposures, a key trait in adaptive immune responses

C. without it, we would need a single gene for every antibody and TCR required to mount all the possible responses needed during our lives

D. interactions with antigens stimulate cells to initate these processes, building the best and strongest response possible to that antigen

E. this is the same DNA altering process that leads to cancer. Understanding it leads to better treatments for cancer.

A.

Question VI. In the typical complement fixation test(CFT), [complete the sentence and assess each subsequent statement(44-55) as either factually correct (= “true”) or incorrect (=“false”)]:


44. a patient positive for the given disease will show an increasein complement in his blood and thus give a positive test.

45. a patient positive for the given disease will show adecrease in complement in his blood and thus give a positivetest.


46. antibody-sensitized sheep red blood cells (sRBC) lyse if thepatient has antigen being tested for.

47. sRBC lyse if the patient has antibody being tested for.


48. sRBC settle to the bottom if the patient has antibody beingtested for.


49. sRBC settle to the bottom if the patient has antigen beingtested for.


50. tested patients have equal amounts of complement in theirserum.


51. tested patients differ in the amount of complement naturallyfound in their serum.

52. the amount of complement in the patient's blood is measuredas an indicator for the presence of infection.


53. a patient's serum is heated to destroy complement and ameasured amount of complement and antigen is added.


54. a patient's serum is heated to destroy complement and ameasured amount of complement and antibody is added.


55. a patient's serum is heated to destroy antibodies and ameasured amount of complement and antibody is added.

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