PAT 20A/B Study Guide - Final Guide: Pulmonary Wedge Pressure, Central Venous Pressure, Neurogenic Shock

• School: Ryerson University
• Department: Pathotherapeutics
• Course: PAT 20A/B
• Professor: Leo Michelis

Document Summary

Shock caused by maldistribution of blood flow ( not getting where it needs to be) Cause: o(cid:272)(cid:272)u(cid:396)s (cid:449)he(cid:374) the (cid:373)yo(cid:272)a(cid:396)diu(cid:373) (cid:272)a(cid:374)"t (cid:272)o(cid:374)t(cid:396)ast suffi(cid:272)ie(cid:374)tly to (cid:373)ai(cid:374)tai(cid:374) ade(cid:395)uate (cid:272)a(cid:396)dia(cid:272) output. Stroke volume and the hea(cid:396)t (cid:272)a(cid:374)"t eje(cid:272)t a(cid:374) ade(cid:395)uate (cid:448)olu(cid:373)e of blood with each contraction. Systolic or diastolic dysfunction results in compromised. Systolic dysfunction inability of the heart to pump blood forward (e. g. myocardial infarction, cardiomyopathy) Diastolic dysfunction inability of the heart to fill during diastole (e. g. pericardial tamponade) Dysrhythmias: (cid:373)yo(cid:272)a(cid:396)dial (cid:272)o(cid:374)t(cid:396)a(cid:272)tility can result in myocardial hypoxia. Blood backs up behind the weakened left ventricle. Hr to compensate for the drop in stroke volume: to maintain cardiac output. As a result of stroke volume: (cid:272)o(cid:396)o(cid:374)a(cid:396)y artery perfusion, (cid:272)ollate(cid:396)al (cid:271)lood flo(cid:449) General manifestations of shock : co. 2: u(cid:396)i(cid:374)e output ( b/c of sodium & water retention, hypotension (cid:271)/(cid:272) the(cid:396)e is(cid:374)"t e(cid:374)ough (cid:272)i(cid:396)(cid:272)ulati(cid:374)g (cid:271)lood, rr, (cid:396)apid shallo(cid:449) (cid:271)(cid:396)eaths, hr, bp, anxious, dizzy, faint, anxiety, confusion, agitation arrhythmias, tachycardia.