PATH 3610 Study Guide - Final Guide: Acute Respiratory Distress Syndrome, Peptic Ulcer, Alveolar Cells

• School: University of Guelph
• Department: Pathology
• Course: PATH 3610
• Professor: Rob Foster

Document Summary

Pathogenesis opportunistic pathogens overcome lung defense due to a. Replace function: ? combination of host, environment and pathogen factors (overcome mucocilary clearance, innate defense proteins and macrophages, recruited leukocytes and acquired immunity) causes inflammation of the bronchi. Inflammation of the airway leads to pulmonary edema, increased liquid in the lungs decreases ventilation and interferes with surfactant to decrease lung compliance. Have local signs such as coughing and expiratory dyspnea, and systemic signs like fever, increased hr, ect. Reduce workload: don"t exercise, or do anything that would increase granuloma (cid:498)tubercle(cid:499) lesion, controlled with t cells. Infiltration with mycobacterium tuberculosis, a facultative pathogen that lives inside alveolar macrophages. Inflamation of the bronchioles and damage to the alveoli. ,causes coughing, fever. Pathogenesis disease that occurs due to other diseases, which cause injury to the. This leads to acute hyaline membrane formation, subacute type ii pneumocyte proliferation and eventual interstitial fibrosis. Also neonatal lung injury due to non- functional surfactant system.