ANP 1106 Study Guide - Final Guide: Ataxia, Corticobulbar Tract, Striatum

• School: University of Ottawa
• Department: Anatomy and Physiology
• Course: ANP 1106
• Professor: Jacqueline Carnegie

Document Summary

Paralysis is different in character depending on which motor neuron effected. The cortex sends direct (corticospinal) and indirect (corticobulbar/bulbospinal) projections to the spinal cord: paralysis. Movement is initiated in the basal ganglia which then projects to the motor cortex aka lesions: akinesia. Movement is regulated by the cerebellum which then projects to the motor cortex lesions: dyskinesia (disruption of movement competence) The cerebellum reads the information given and revises/corrects the projection. Without the cerebellum a very sloppy set of instruction is outputted. Therefore, the motor cortex executes the movement, but the cerebellum regulates it. The brain stem nuclei do not produce voluntary movements on their own. Which means if the motor cortex is damaged then you cannot control voluntary movement (paralysis). Therefore, they are not executers of movement but ------ Primary motor cortex: the precentral gyrus (4) of frontal lobe *note its relationship to the primary somatosensory cortex (3,2,1). Controls activities of small fragmented muscle grouped movements.