PHTY206 Lecture Notes - Lecture 14: Bursitis, Proprioception, Current History

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Physical Examination of the Hip
Learning Outcomes
o Describe the clinical presentation and pathology of common disorders of the hip
o List the various anatomical sources that may contribute to symptoms in the hip region
o Recognize clinical signs of a joint versus a muscle disorder in the hip
**Note: revise functional anatomy of the hip and groin region
Where is the pain coming from?
Common Causes of hip pain
Anterior Pain
o Hip Osteoarthritis (OA)
OA is the soial joits age related respose to abnormal loading or abnormal
mechanics caused by a traumatic incident or repetitive microtrauma
Trauma causes cartilage degradation, initiating an inflammatory process which
may lead to further cartilage damage.
OA can affect the whole joint organ (Brandt et al. 2009)
Cartilage degradation
Progressive damage to subchondral bone
Microfractures
Hypertrophic (osteophytic) and atrophic bone response
Bone necrosis and periostitis
Inflammation of the synovium and synovial lining thickening
OA characteristics lead to pain & stiffness, to functional limitations and
subsequent muscle changes
X-ray Diagnosis
X-ray Diagnosis Kellgren & Lawrence scores 1-4
Subchondral bone sclerosis
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Narrowing of joint space
Formation of osteophytes at joint margin
Subchondral cysts
Superior migration of the femoral head secondary to articular
cartilage loss.
Diagnostic Imaging for Hip OA
Magnetic Resonance Imagining (MRI)
Ultrasound
Scintigraphy
CT arthrography
Used to identify early microscopic cartilaginous changes, bone marrow lesions
and oedema or other joint changes
Hip OA vs. RA
OA
RA
Local, usually affecting single joint,
develops gradually
Morning stiffness < 1 hour
Pain and stiffness in the affected joint
X-rays - single hip joint usually affected,
subchondral bone sclerosis and osteocytes
Autoimmune systemic disease, affecting multiple
joints with pain, stiffness and swelling (e.g. hands)
Periodic flare up and remission
Morning stiffness > 1 hour
Fatigue, loss of appetite
X-rays - may have bilateral hip joint signs,
demineralisation or femoral head, articular erosion
Predisposing factors of OA
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Risk Factors
Age - increased risk in >55yr olds, with further increased risk in 65 - 80 year
old
Gender/sex - increased risk in women
Genetic/familial predisposition
Ethnicity or race - lower incidence in Asian cultures than white Caucasian.
Very low incidence in Koreans.
Morphological abnormalities/developmental disorders
Leg length difference
History of lower limb or hip trauma
Occupational factors
Manual labour/physical stress work
Sporting activities
Increased weight or obesity (BMI) (questionable for development of OA but
yes for progression)
American College of Rheumatology Clinical Diagnostic Criteria
Patient Interview
Main problem
Joint pain & stiffness
Pain usually related to movement e.g.: walking, driving, stair climbing,
gardening
Stiffness in early morning or after rest which eases with movement
Difficulty putting socks and shoes on
Description of symptoms
deep ahe or disofort ith sharp staig pai
joint stiffness, restricted mobility
Area of symptoms
Reporting pain anterior/posterior or lateral hip region +/- referral to
thigh/knee
Behaviour of symptoms
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Document Summary

2009: cartilage degradation, progressive damage to subchondral bone, microfractures, hypertrophic (osteophytic) and atrophic bone response, bone necrosis and periostitis. Inflammation of the synovium and synovial lining thickening: (cid:1372) oa characteristics lead to pain & stiffness, to functional limitations and subsequent muscle changes, x-ray diagnosis, x-ray diagnosis kellgren & lawrence scores 1-4. Subchondral bone sclerosis: narrowing of joint space. Superior migration of the femoral head secondary to articular cartilage loss: diagnostic imaging for hip oa, magnetic resonance imagining (mri, ultrasound. Scintigraphy: ct arthrography, (cid:1372) used to identify early microscopic cartilaginous changes, bone marrow lesions and oedema or other joint changes, hip oa vs. ra. Leg length difference: morphological abnormalities/developmental disorders, history of lower limb or hip trauma, occupational factors, manual labour/physical stress work. Increased weight or obesity (bmi) (questionable for development of oa but yes for progression: american college of rheumatology clinical diagnostic criteria, patient interview, main problem.

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