ACBS 457 Lecture Notes - Lecture 5: Bluetongue Disease, African Trypanosomiasis, Ceratopogonidae

• School: University of Arizona
• Department: Animal & Comp Biomed Sciences
• Course: ACBS 457
• Professor: Dr. Riehle

Document Summary

Test covers: other mosquito borne diseases --> lice. The host cell lysosomes migrate and fuse with the parasitophorus vacuole. The lysosome content is released into the vacuole, however the parasite is not altered. Most amastigotes become attached to the membrane of the vacuole by their posterior portion and the amastigote divides multiple times. The parasitophorus vacuole can fill the entire cytoplasm of the host cell and eventually explode to infect again. Blood meal of infected vertebrate host and ingest blood containing infected macrophages with amastigote forms. In new environment amastigotes form clusters (cid:862)(cid:374)est (cid:272)ells(cid:863) e(cid:374)(cid:272)losed (cid:271)y peritrophic matrix. Amastigotes pass through thoracic mid-gut to abdominal mid-gut. The interior form of the peritrophic matrix breaks down and parasites are released to the mid- gut of the insect. Promastigotes divide by binary fission and attach at the mid-gut. When the parasites detach from the epithelium the promastigotes migrate toward the.