21. Aerobic training would decrease submaximalheart rate, which is beneficial for aerobic performance because
a. it saves energy used by heart beats.
b. it gives longer time for ventricular filling.
c. a and b.
d. None of above.
22. Aerobic training would ________ anaerobicpower during maximal exercise.
a. delay onset of
b. facilitate onset of
c. rely on
23. Compared to untrainedindividuals, trained endurance athletes have
a. higher maximal heartrates.
b. higher resting cardiacoutputs.
c. higher resting heartrates.
d. higher maximal strokevolumes.
24. What is the likely outcome oflow volume, low intensity training?
a. increased aerobiccapacity
b. increased anaerobiccapacity
c. increased aerobic andanaerobic capacity
d. little improvement in eitheraerobic or anaerobic capacity
25. Which of the followingstatements explaining the increase in SV after aerobic training iscorrect?
a. reload, contractility, andafterload increase
b. preload, contractility, andafterload decrease
c. preload and contractilityincrease, afterload decreases
d. preload decreases,contractility and afterload increase
26. When motor units contract moresynchronously,
a. muscles cannot sustainsteady forces anymore
b. muscles increase the rate offorce development
c. the ability to generateforce is impeded
d. a single motor neuron nowinnervates several motor units
27. In general, whole-muscle hypertrophy in humans most likely resultsfrom
a. mostly fiberhyperplasia
b. entirely fiberhyperplasia
c. mostly fiberhypertrophy
d. equal contributions of fiberhypertrophy and fiber hyperplasia
28. Overload can be achieved by
a. increasing intensity.
b. increasing recovery time.
c. a certain type of training.
d. All of above.
29. Resistance training increases forceproduction by
a. neural adaptation followed by intramuscular adaptation.
b. intramuscular adaptation followed by neural adaptation.
c. simultaneous neural and intramuscular adaptation.
30. Training causesan increase in stroke volume due to all of the following except
a. increased filling time.
b. increased left ventricular volume.
c. increased cardiac and arterial stiffness.
d. improved intrinsic contractile function.
31. The risk of DOMS after a giventraining session can be reduced by which trainingstrategy?
a. train at high intensity withboth resistance and anaerobic training components
b. train with primarily intenseeccentric exercise
c. reduce eccentric componentof muscle action
d. employ more downhillexercise in the training program
32. After anaerobic training, thecross-sectional areas of ______ are larger than before thetraining.
a. type I fibers
b. type II fibers
c. type I and type IIfibers
33. Aerobic training would cause __________cardiac hypertrophy, which is increased ______________.
a. concentric, the diameter of left ventricle.
b. eccentric, the diameter of left ventricle.
c. concentric, the wall thickness.
d. eccentric, the diameter and wall thickness of leftventricle.
34. InFartlek training, ___________ is changed based on
21. Aerobic training would decrease submaximalheart rate, which is beneficial for aerobic performance because
a. it saves energy used by heart beats.
b. it gives longer time for ventricular filling.
c. a and b.
d. None of above.
22. Aerobic training would ________ anaerobicpower during maximal exercise.
a. delay onset of
b. facilitate onset of
c. rely on
23. Compared to untrainedindividuals, trained endurance athletes have
a. higher maximal heartrates.
b. higher resting cardiacoutputs.
c. higher resting heartrates.
d. higher maximal strokevolumes.
24. What is the likely outcome oflow volume, low intensity training?
a. increased aerobiccapacity
b. increased anaerobiccapacity
c. increased aerobic andanaerobic capacity
d. little improvement in eitheraerobic or anaerobic capacity
25. Which of the followingstatements explaining the increase in SV after aerobic training iscorrect?
a. reload, contractility, andafterload increase
b. preload, contractility, andafterload decrease
c. preload and contractilityincrease, afterload decreases
d. preload decreases,contractility and afterload increase
26. When motor units contract moresynchronously,
a. muscles cannot sustainsteady forces anymore
b. muscles increase the rate offorce development
c. the ability to generateforce is impeded
d. a single motor neuron nowinnervates several motor units
27. In general, whole-muscle hypertrophy in humans most likely resultsfrom
a. mostly fiberhyperplasia
b. entirely fiberhyperplasia
c. mostly fiberhypertrophy
d. equal contributions of fiberhypertrophy and fiber hyperplasia
28. Overload can be achieved by
a. increasing intensity.
b. increasing recovery time.
c. a certain type of training.
d. All of above.
29. Resistance training increases forceproduction by
a. neural adaptation followed by intramuscular adaptation.
b. intramuscular adaptation followed by neural adaptation.
c. simultaneous neural and intramuscular adaptation.
30. Training causesan increase in stroke volume due to all of the following except
a. increased filling time.
b. increased left ventricular volume.
c. increased cardiac and arterial stiffness.
d. improved intrinsic contractile function.
31. The risk of DOMS after a giventraining session can be reduced by which trainingstrategy?
a. train at high intensity withboth resistance and anaerobic training components
b. train with primarily intenseeccentric exercise
c. reduce eccentric componentof muscle action
d. employ more downhillexercise in the training program
32. After anaerobic training, thecross-sectional areas of ______ are larger than before thetraining.
a. type I fibers
b. type II fibers
c. type I and type IIfibers
33. Aerobic training would cause __________cardiac hypertrophy, which is increased ______________.
a. concentric, the diameter of left ventricle.
b. eccentric, the diameter of left ventricle.
c. concentric, the wall thickness.
d. eccentric, the diameter and wall thickness of leftventricle.
34. InFartlek training, ___________ is changed based on
