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In your own words, What are the circumstances of disease transmission in the three cases below:

1) Congenital Visceral Leishmaniasis

2) Leishmania infantum and Human Visceral Leishmaniasis, Argentina

3) African trypanosomiasis in travelers returning to the united kingdom

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Jean Keeling
Jean KeelingLv2
28 Sep 2019

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Related questions

In your own words please follow the links bellow and answer the question be as detailed please.

https://www.cdc.gov/mmwr/preview/mmwrhtml/mm5110a3.htm

https://wwwnc.cdc.gov/eid/article/18/2/pdfs/11-0924.pdf

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3373117/

Briefly, summarize and compare the two case studies on leishmaniasis and Trypanosoma cruzi. What were the circumstances of disease transmission in the three cases, how was the infection diagnosed in each case and what methods were used for diagnosis? What new information is contained in the cases that are not found in the CDC website?

In your own word, Compare the pathogenesis of visceral leishmaniasis with Chagas disease (be specific and answer in complete sentences).

Visceral leishmaniasis is transmitted through the bite of a female sand fly. The sand fly bites the human and injects the promastigote into the blood known as the infection stage. Next promastigotes are phagocytized by the macrophages and transform into amastigotes inside the macrophages. The amastigotes then multiply inside the cells and then is picked up by a sand fly biting the human infected where the amastigotes transform into promastigotes inside the gut of the fly and divide. The process starts all over.

Chagas disease starts with the triatomine bug biting a human and passing the metacyclic trypomastigotes through their feces into the bite wound known as the infection stage. Once inside the host they penetrate cells and turn into amastigotes where they multiply by binary fission. The intracellular amastigotes transform into trypomastigotes and burst the cell entering the blood stream or they can infect other cells and start the transformation to amastigotes all over. Once they enter the blood stream the trypomastigotes can be picked up by another triatomine bug when they bite the human. Once inside the gut of the bug they turn into epimastigotes and multiply before turning back in to metacyclic trypomastigotes. The process starts all over.

short answers( Just few words) Basic bio, answer them all and I will rate you

1-What is the most apical stratum of the epidermis? Visceral? Which layer is not present in thick skin?

2-Thick skin has five strata and thin has four, but this is not the primary reason why thick skin is so much thicker than thin skin. What is the primary reason?

3-What is the most abundant cell in the epidermis of your skin? What other cells are located in the epidermis? In which stratum are the cells you identified located?

4-How long does it take a keratinocyte produced in the stratum germinativum to be sloughed off the surface of the epidermis?

5-What does it mean that the epidermis is avascular? Virtually all cells of the human body are within 20 ร‚ยตm of a capillary. Do some of the cells of the epidermis fly in the face of this general rule?

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