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29 Oct 2018

Draw and describe the mechanisms that can convert a proto-oncogene into an oncogene.

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Sixta Kovacek
Sixta KovacekLv2
1 Nov 2018

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Related questions

One way that RNA tumor viruses can cause cancer upon infection is by commandeering a host gene and transcribing it under a strong promoter in the viral LTR region of the viral genome.

a. What evidence suggests that a proto-oncogene like src is actually a normal cellular gene rather than a gene that has been inserted into the germ line by an infecting retrovirus?

b. What is the normal function of src in the cell?

c. Describe the regulation of this proto-oncogene include how the dysfunction of this proto-oncogene leads to cancer.

QUESTION 7

What is the purpose of gap junctions?

A. To prevent the leakage of extracellular fuid.

B. To provide cytoplasmic channels between adjacent plant cells.

C. To fasten cells together.

D. To provide cytoplasmic channels between adjacent animal cells.

QUESTION 8

During which phase of the cell cycle do homologous pairs of chromosomes line up on the metaphase plate?

A. Metaphase III

B. Metaphase I

C. Metaphase

D. Metaphase II

QUESTION 9

Which one of the following is NOT a mechanism that contributes to genetics variation?

A. Random pairing of homologous chromosomes

B. Random fertilization

C. Crossing over

D. Independent assortment of chromosomes

QUESTION 10

Generally speaking, how does a proto-oncogene become an oncogene?

A. The proto-oncogene is an inactive precursor protein that is cleaved to form the mature oncogene.

B. The proto-oncogene is phosphorylated, altering the function to that of an oncogene.

C. Proto-oncogenes can never become oncogenes. These are two completely different things.

D. The proto-oncogene is mutated, altering the function to that of an oncogene.

QUESTION 11

During which process are gametes (sperm and egg) produced?

A. Mitosis

B. Budding

C. Binary Fission

D. Meiosis

1. this is a tumor suppressor protein that interacts with cyclin-kinases when there is DNA damage.

p21

S phase inhibitor

p53

RB

2. this protein is a transcriptional activator AND a proto-oncogene:

RB

wee1

E2F

p53

3. to help cure a proto-oncogene cancer one would try__

antibodies only

antibodies and antisense RNA

overexpression

gel shift

4. to show that a virus makes proteins to interact with p53 protein, one would do_

gel shift

NPA

foot print

co-immunoprecipitation

5. overexpression or activation of ____ leads to immortal cancer cells

Wee1

CAK

telomerase

E2F

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